Dr. Ellis R. Levin

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  www.scire-lb.org/services/investigator/index.htm - [Cached Version]
  Published on: 10/11/2008    Last Visited: 10/14/2008  

  Ellis Levin, M.D.

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  www.ssr.org/07Schedule.html - [Cached Version]
  Last Visited: 8/18/2007  

  Ellis R. Levin, M.D. (University of California, Irvine, Irvine, California): Extra-nuclear estrogen receptor signaling.

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  www.ssr.org/07SciSessions.html - [Cached Version]
  Last Visited: 8/18/2007  

  Speakers: Stephen Hammes, M.D., Ph.D. (University of Texas Southwestern Medical Center, Dallas, Texas): Transcription-Independent Steroid Signaling in the Ovary; Peter Thomas, Ph.D. (University of Texas at Austin, Austin, Texas): G Protein Signaling and Functions of the Novel Progesterone Membrane Receptors in Reproductive Tissues; Ellis R. Levin, M.D. (University of California, Irvine, Irvine, California): Extra-nuclear estrogen receptor signaling.

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  www.ssr.org/07ProgramHighlights.html - [Cached Version]
  Last Visited: 3/8/2007  

  Speakers: Stephen Hammes, M.D., Ph.D. (University of Texas Southwestern Medical Center, Dallas, Texas): Transcription-Independent Steroid Signaling in the Ovary; Peter Thomas, Ph.D. (University of Texas at Austin, Austin, Texas): G Protein-Coupled Progesterone Membrane Receptor Signaling in Reproductive Tissues; Ellis R. Levin, M.D. (University of California, Irvine, Irvine, California): Novel Functions of Membrane Estrogen Receptor Signaling.

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  2005 Annual Meeting - American Association for Cancer... - [Cached Version]
  Published on: 9/6/2002    Last Visited: 12/8/2004  

  Ellis R. Levin, University of California Irvine, Long Beach, CA

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  AWIS::Voice::The Washington Wire - [Cached Version]
  Published on: 8/1/2004    Last Visited: 8/25/2005  

  Dr. Ellis Levin, a professor of medicine, biochemistry and pharmacology at University of California at Irvine, and colleagues at the Long Beach Veterans Administration Medical Center and Georgetown University Lombardi Cancer Center have identified how the healthy BRCA1 gene prevents the growth and survival of breast cancer cells.It may provide further evidence as to how estrogen helps activate a disease that afflicts thousands of women each year.Levin said that the findings give further insight into how estrogen can interact with mutant BRCA1 to promote the development of breast cancer."This discovery is important", he added, "because women who take estrogen supplements for more than five years to treat menopause symptoms increase their chances of developing breast cancer by about 25 percent."

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  Apria - resources - News - [Cached Version]
  Published on: 12/28/2003    Last Visited: 7/28/2004  

  Dr. Ellis Levin, a professor of medicine, biochemistry and pharmacology at UCI, and colleagues at the Long Beach Veterans Administration Medical Center and Georgetown University Lombardi Cancer Center have identified how the healthy BRCA1 gene prevents the growth and survival of breast cancer cells.
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  Levin said that the findings give further insight into how estrogen can interact with mutant BRCA1 to promote the development of breast cancer.This discovery is important, he added, because women who take estrogen supplements for more than five years to treat menopause symptoms increase their chances of developing breast cancer by about 25 percent.
  
  "The therapeutic goal is to develop estrogen inhibitors that would prevent these types of undesirable effects, yet preserve the positive effects that prevent osteoporosis or hot flashes.Alternatively, enhancing or restoring normal BRCA1 protein function is another approach to consider in women with such BRCA1 mutations," said Levin, who also is chief of endocrinology at UCI and at the VA Medical Center.

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  Artemis -- September 2004 - [Cached Version]
  Published on: 9/1/2004    Last Visited: 3/31/2008  

  Dr. Ellis Levin, a professor of medicine, biochemistry and pharmacology at UCI, and colleagues at the Long Beach Veterans Administration Medical Center and Georgetown University Lombardi Cancer Center have identified how the healthy BRCA1 gene prevents the growth and survival of breast cancer cells.
  ...
  Levin said that the findings give further insight into how estrogen can interact with mutant BRCA1 to promote the development of breast cancer.This discovery is important, he added, because women who take estrogen supplements for more than five years to treat menopause symptoms increase their chances of developing breast cancer by about 25 percent.
  
  "The therapeutic goal is to develop estrogen inhibitors that would prevent these types of undesirable effects, yet preserve the positive effects that prevent osteoporosis or hot flashes.Alternatively, enhancing or restoring normal BRCA1 protein function is another approach to consider in women with such BRCA1 mutations," said Levin, who also is chief of endocrinology at UCI and at the VA Medical Center.

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  Biological Psychology Links - [Cached Version]
  Published on: 4/30/2002    Last Visited: 5/2/2002  

  "There's been a tremendous turn-around by a number of prominent people in the field who are now looking at this concept very seriously," says senior author Ellis R. Levin, chief of endocrinology and metabolism at the Long Beach Veterans Affairs Medical Center and vice chairman for research in the department of medicine at the University of California, Irvine.The Scientist 15[8]:15, Apr. 16, 2001 © Copyright 2001, The Scientist, Inc.All rights reserved.See also: Chapter 5: Hormones and the BrainPosted: 10.20.2001
  
  Ring-breaker drives dove love

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  Breast Cancer: Bidirectional signaling between EGFR... - [Cached Version]
  Published on: 5/6/2003    Last Visited: 5/11/2003  

  This results from signal transduction enacted by the plasma membrane tyrosine kinase growth factor receptor, leading to 1) phosphorylation and activation of the nuclear ER, and 2) phosphorylation of coregulator proteins," wrote E.R. Levin and colleagues, University of California Irvine, Long Beach Veterans Affairs Medical Center.
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  Levin and colleagues published their study in Molecular Endocrinology (Bidirectional signaling between the estrogen receptor and the epidermal growth factor receptor.Mol Endocrinol, 2003;17(3):309-317).
  
  For more information, contact E.R. Levin, University of California Irvine, Long Beach Veterans Affairs Medical Center, Medical Service 111I, Division of Endocrinol, Long Beach, CA 90822, USA.

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