Last Update

2015-10-02T00:00:00.000Z

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Wrong William Goodman?

Dr. William G. Goodman

Clinical Research Director

Amgen Inc.

Direct Phone: (805) ***-****       

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Amgen Inc.

One Amgen Center Drive

Thousand Oaks, California 91320

United States

Company Description

Amgen is committed to unlocking the potential of biology for patients suffering from serious illnesses by discovering, developing, manufacturing and delivering innovative human therapeutics. This approach begins by using tools like advanced human genetics ... more

Find other employees at this company (23,331)

Background Information

Employment History

Professor of Medicine, Director

UCLA

Affiliations

Member, Editorial Boards
BioDiscussion

Member
American Society for Bone and Mineral Research

Member, Editorial Boards
Journal of the American Society of Nephrology

Member
American Society of Nephrology

Member
International Society of Nephrology

Member of the Scientific Advisory Council
National Kidney Foundation of Southern California Inc

Education

MD

medical degree

The Ohio State University College of Medicine

Web References (54 Total References)


Hidden Dangers - Elements Of Health

elementsofhealth.webs.com [cached]

What awakened doctors to this connection was a study conducted by Dr. William Goodman of the University of California, Los Angeles.

Goodman and fellow UCLA anthropologist Dr. Isidro Salusky used powerful new imaging technology to scan the arteries of 39 young dialysis patients.


Hidden Dangers - Elements Of Health

elementsofhealth.webs.com [cached]

What awakened doctors to this connection was a study conducted by Dr. William Goodman of the University of California, Los Angeles.

Goodman and fellow UCLA anthropologist Dr. Isidro Salusky used powerful new imaging technology to scan the arteries of 39 young dialysis patients.


Mednet - CME, CHE | Management of Bone and Mineral Abnormalities in Chronic Kidney Disease

www.mednet.ca [cached]

“This decline occurs before there is any elevation in serum phosphorus,� Dr. William Goodman, Professor of Medicine, David Geffen School of Medicine, noted. Abnormalities in vitamin D metabolism in turn adversely affect intestinal calcium transport. Thus, in Dr. Goodman’s view, elevations in PTH in early-stage CKD is an appropriate physiological response that helps maintain serum calcium levels. In both healthy individuals as well as in patients with mild to moderate CKD, phosphorus loading impairs the kidney’s production of 1,25-dihydroxyvitamin D3, while restricting phosphorus will increase its production.

The literature suggests that even if phosphorus level is normal, restricting phosphorus in the diet or through the use of phosphate binders will drive up 1,25-dihydroxy-vitamin D3 production and help prevent bone disease. Increases in 1,25-dihydroxyvitamin D3 levels correspond with subsequent reductions in plasma PTH levels, “so it is the change in circulating 1,25-dihydroxyvitamin D3 levels that mediates the reduction in PTH, not phosphorus per se,� Dr. Goodman indicated.
He also argued that patients with earlier-stage CKD are fundamentally different than those on dialysis, largely because they still have residual kidney function and they can excrete phosphorus. In patients who have mild to moderate CKD, serum phosphorus levels are usually normal, while serum calcium levels are typically in the lower range of normal, he added; and the fact that patients with mild CKD excrete relatively little calcium is a sign that the kidney is trying to conserve calcium. “I’m not arguing with the fact that phosphate restriction and phosphate binders are appropriate when serum phosphate levels are elevated,� Dr. Goodman stated.
...
Data supporting a non-calcium-based phosphate binder strategy are limited but one prospective studyâ€"the “treat-to-goalâ€� trialâ€"showed that vascular calcification did not worsen over one year of follow-up in dialysis patients treated with sevelamer, whereas it did progress in patients treated with a calcium phosphate binder. “Theoretically, the use of lanthanum carbonate might have a similar beneficial effect because it is also calcium-free,â€� Dr. Goodman indicated, although this has not yet been demonstrated in a prospective trial.
However, he stated that the choice of a phosphate binding agent could influence progression of calcification once it is present, even though it is not clear how vascular calcification develops in the first place.
...
“Your choice of phosphate binder is going to be influenced very significantly by other biochemical abnormalities,� Dr. Goodman cautioned.
...
In an interview, Dr. Goodman also suggested that this agent might be equivalent in potency to the old aluminum-containing binders and more potent than calcium carbonate nor calcium acetate.


BioDiscussion

www.biodiscussion.com [cached]

William Goodman, MD BioDiscussion

...
William Goodman, MD Professor of Medicine Director, Bone, Histology and Molecular Bone Labs of the CRC UCLA School of Medicine
...
William Goodman, MD has documented that he/she has nothing to disclose.


BioDiscussion

www.biodiscussion.com [cached]

Panelists: William Goodman, MD; Peter McCullough, M.D., M.P.H.; Charles Nolan, MD

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