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Wrong Vladimir Svetlov?

Vladimir Svetlov

Senior Research Scientist

NYU

HQ Phone:  (212) 263-7300

Email: v***@***.org

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NYU

550 First Avenue

New York City, New York,10016

United States

Company Description

NYU Langone Medical Center, a world-class, patient-centered, integrated academic medical center, is one of the nation's premier centers for excellence in clinical care, biomedical research, and medical education. Located in the heart of Manhattan, NYU Langone ... more.

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Background Information

Employment History

Postdoc-Factotum

The Ohio State University


Web References(11 Total References)


engineering.curiouscatblog.net

Jam the switch and you might prevent infection, said Vladimir Svetlov, a microbiology research associate at Ohio State University and one of a group of scientists who defined the structure of a protein that appears to be the key.


www.biocompare.com [cached]

Vladimir Svetlov, PhD
Research Associate II Department of Microbiology Ohio State University


www.omniomix.com

Knowing how disease-causing bacteria, like Yersinia pestis and E. coli, do this may one day help scientists create drugs that control the expression of these genes, thereby making the bacteria harmless, said Vladimir Svetlov, a study co-author and a research associate in microbiology at Ohio State University."In contrast to NusG, which is always active, RfaH is usually inactive, because the part of the protein that is needed to activate gene expression is typically masked," Svetlov said."Cells usually don't die when RfaH use changes," Svetlov said.Svetlov and Artsimovitch conducted the study with Georgy Belogurov, a postdoctoral research associate in microbiology at Ohio State, and with researchers from the University of Alabama at Birmingham, the Howard Hughes Medical Institute and the University of Texas Southwestern Medical Center in Dallas.Contact: Vladimir Svetlov, (614) 688-3561; Svetlov.1@osu.edu


www.eurekalert.org

Contact: Vladimir SvetlovSvetlov.1@osu.edu614-688-3561Ohio State University Bacteria control how infectious they become, study findsCOLUMBUS, Ohio - The results of a new study suggest that bacteria that cause diseases like bubonic plague and serious gastric illness can turn the genes that make them infectious on or off.Knowing how disease-causing bacteria, like Yersinia pestis and E. coli, do this may one day help scientists create drugs that control the expression of these genes, thereby making the bacteria harmless, said Vladimir Svetlov, a study co-author and a research associate in microbiology at Ohio State University."In contrast to NusG, which is always active, RfaH is usually inactive, because the part of the protein that is needed to activate gene expression is typically masked," Svetlov said."Cells usually don't die when RfaH use changes," Svetlov said.Svetlov and Artsimovitch conducted the study with Georgy Belogurov, a postdoctoral research associate in microbiology at Ohio State, and with researchers from the University of Alabama at Birmingham, the Howard Hughes Medical Institute and the University of Texas Southwestern Medical Center in Dallas.Contact: Vladimir Svetlov, (614) 688-3561; Svetlov.1@osu.edu


www.arn.org [cached]

The letter, by Vladimir Svetlov, a microbiologist at Ohio State University, chides Nature for making such a big deal out of the fact that a pro-ID article was published in a peer-reviewed journal science journal.
"I cannot in all honesty share in the anxiety surrounding publication of a dubious paper on 'intelligent design'—regarded by most scientists as a version of creationism—in a journal with an impact factor of less than one," says Svetlov. "Your News story "Peer-reviewed paper defends theory of intelligent design" (Nature 431, 114; 2004) suggests that getting an intelligent-design paper into a peer-reviewed journal is a huge achievement for creationism." To the contrary, he argues, the real surprise is that ID proponents didn't get such a publication earlier. Why? Because "one can publish just about anything if one goes far enough down the list of impact factors. There are papers all around us containing problems glaring enough to fail their authors in undergraduate midterm exams." Svetlov may not understand why the publication of Meyer's paper was such a big deal. But doing so affirms the likelihood that peer-reviewed journals have published some rubbish—maybe a good deal of it, as Svetlov asserts.


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